Biomedical application of TiO2NPs can cause arterial thrombotic risks through triggering procoagulant activity, activation and aggregation of platelets-Yeeran

Biomedical application of TiO2NPs can cause arterial thrombotic risks through triggering procoagulant activity, activation and aggregation of platelets

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Bian Y, Jin Q, He J, Ngo T, Bae ON, Xing L, Pi J, Chung HY, Xu Y. Biomedical application of TiO2NPs can cause arterial thrombotic risks through triggering procoagulant activity, activation and aggregation of platelets. Cell Biol Toxicol. 2024 Aug 7;40(1):67. doi: 10.1007/s10565-024-09908-y.

Sigma-1 receptor activation attenuates DOX-induced cardiotoxicity by alleviating endoplasmic reticulum stress and mitochondrial calcium overload via PERK and IP3R-VDAC1-MCU signaling pathways

CARD9 protein SUMOylation regulates HOXB5 nuclear translocation and Parkin‐mediated mitophagy in myocardial I/R injury

Sigma-1 receptor activation attenuates DOX-induced cardiotoxicity by alleviating endoplasmic reticulum stress and mitochondrial calcium overload via PERK and IP3R-VDAC1-MCU signaling pathways

CARD9 protein SUMOylation regulates HOXB5 nuclear translocation and Parkin‐mediated mitophagy in myocardial I/R injury

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